Fulltext – Ascochyta blight (Ascochyta rabiei (Pass.) Lab.) of Chickpea (Cicer arietinum L.): Breeding Strategies for Resistance. Ascochyta blight, a fungal disease caused by Ascochyta rabiei (Pass.) Labrousse , is the major constraint for chickpea (Cicer arietinum L.). Ascochyta blight caused by Ascochyta rabiei (Pass.) Labr. is one of the major disease of chickpea worldwide. Since there is report of variable number of races .

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Current status and future strategy in breeding chickpea for resistance to biotic and abiotic stresses. Relative importance of various biotic and abiotic stresses resistance in chickpea.

Classification of pathotypes of A. Cookies mentioned in the last point are Google analytics cookies that are IP anonymized which means that we cannot trace single users. International Journal of Plant Breeding and Genetics, 5: Specimen record 4.

Furthermore, the number of EST and genomic sequences available in databases is growing rapidly especially from genome sequencing projects e.

Ascochyta blight resistance depends more on the genetic background than leaf type. It is important to note, however, that complete resistance to A. Heritability of some important plant rahiei, their relationships with plant yield and inheritance of Ascochyta blight resistance in chickpea Cicer arietinum L. The blight of chickpea Cicer aritinum. Mapping of gene-specific markers on the genetic map of chickpea Cicer arietinum L.


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The origin of chickpea Cicer arietinum L. These assays were conducted by artificial inoculation either in the field or under plastic-house conditions. High humidity and temperature promote sporulation.

Didymella rabiei in the Pacific Northwest raibei the United States. Genetic and pathogenic diversity within Ascochyta rabiei Pass. Pathotype-specific genetic factors in chickpea Cicer arietinum L.

Identification of genetic diversity of Ascochyta blight [ Ascochyta rabiei Pass. In this study, initially locus specific primer pairs were designed for 22 microsatellite containing regions from C.

Microsatellite markers for molecular breeding. Tewari and Pandey reported that AB resistance in chickpea was governed by two recessive dabiei through additive gene action.

Conidia are dispersed to neighboring plants through rain splashing Armstrong et al. Specimen record 8. Content is available for non-profit, educational use under our Copyright Agreement.

Low temperature and a relatively long incubation period are required for sexual reproduction in most Ascomycetes. Khune, The Coelomycetes of India: Production of chickpea contributed to agricultural sustainability through N 2 -fixation and by being a rotation crop. Later, they develop a brown margin with a white center and split longitudinally. Localization of resistance genes for fusarium wilt races 4 and 5. In desi cultivars, the resistance was governed by a single dominant gene Taleei et al.


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The families of pathogenesis-related proteins, their activities and comparative analysis of PR-1 type proteins. Breeding for Resistance to Ascochyta Blight in Chickpea. In the second method, the number of leaves showing symptoms or wilting and the total number ascochyyta leaves on each plant were counted and the percentage of infected leaves for each plant calculated Kanouni et al. Preliminary investigation of QTLs associated with seedling resistance to ascochyta blight from Cicer echinospermuma wild relative of chickpea.

Chickpea Cicer arietinum L. Ascochyta rabiei form typical appressoria associated with stomatal penetration Illarslan and Dolar, Didymella rabiei growing on chickpea.

Didymella rabiei – Wikipedia

Antibiosis mechanism of resistance to pod borer, Helicoverpa armigera in wild relatives of chickpea. Genetic diversity studies in a crop are important for various aspects such as management of genetic resources, identification of duplicate accessions in the germplasm ascochhyta in applied breeding programs. Marker Assisted Selection MAS can be applied effectively using DNA markers linked to resistance genes for accelerating disease resistance breeding programs.

Infection and host resistance mechanisms.

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